Intestinal parasites have penetrated the human brain and lived there for years

Neurocysticercosis, the most annoying way to infect your brain with tapeworms, has fallen to the ground. . The commotion woke her husband, who found her husband on the floor shivering and saying "nonsense." He was transferred to Massachusetts General Hospital

There, doctors noted that the man had a two-minute tonic-clonic seizure (Grand Mall) in which he lost consciousness and his muscles contracted sharply. The doctors began the arduous process of trying to collect the errors by running a series of tests and interviewing his family.

In almost all cases, the man was in very good health. He is a father of two children, and has no history of seizures, cardiovascular, respiratory, gastrointestinal, genitourinary, or neurological disorders. His toxicology pages were straightforward. He did not take any prescription or over-the-counter medications. He does not smoke and rarely drinks. There was no evidence of anything happening to him recently that caused an epileptic fit. The man had spent the previous day with his children, then had dinner with his brother, who had reported nothing unusual. The only initial sign of illness was that the man had immigrated to Boston about 20 years ago from a rural area in Guatemala.

They did. They quickly narrowed down the possibilities. Scans showed three calcified lesions in his brain, and doctors diagnosed him with neurocysticercosis. In other words, the larval cysts of the tapeworm migrated to his head several years ago and settled in various parts of his brain. Doctors documented the man's work in a case study published in the New England Journal of Medicine on Thursday, November 11. stomach weakness; This is as raw a disaster as it is serious. The only pork tapeworm usually accumulates in the human intestine, where it can grow an impressive length of 2 to 8 meters. Meanwhile, victims of this worm erupt parasitic eggs in their feces. If this egg-filled feces enter an environment with pigs, the pigs can follow the life cycle of the worm by eating the eggs.

In a pig's stomach, stomach acid causes the eggs to hatch, causing you to lose your bodyguard. They cover and appear as cysts of larvae called neoplastic balls. They can penetrate the intestinal wall and travel through the circulatory system in pigs. Eventually, they burrow into the muscles of the pigs and hide in the form of a cyst - which is usually not bothersome to pigs.

However, if a person eventually eats an uncooked pork containing those larval sacs, the life cycle continues. . In the human digestive system, the worm emerges from its cystic form and enters its hook and four suckers in the human upper intestine. There, he can die happily for years, elongating his ribbon-like body, and laying more eggs. And the life cycle begins again.

Everything is going right, but only when a human - not a pig - finally eats the eggs of the worm. This can happen in a disgusting scenario where a person infected with tapeworms is in poor health and also prepares food. In other words, tapeworms contaminate food with feces. In this case, eggs come out like pigs in the human stomach. Larval sacs can end up in human muscles (worm sacs), but they may also migrate to the eyes and brain (neurocysticercosis). This is a dead end for worms and can be a huge problem for humans. At first, it quietly lurks like a live worm, causing little immune response and therefore no symptoms. This step may take years. But over time, the cyst dissolves and leaks a fluid that alerts the immune system that a parasite is present and that there is a strong reaction. The cyst degenerates further and forms a knot in the brain. Eventually the nodule becomes a calcified granuloma. Seizures are associated with inflammatory responses associated with late-stage calcification, and neurocysticercosis is the most common parasitic infection of the human brain and can cause headaches, confusion, balance problems, seizures, and even death. It is also the most common cause of acquired epilepsy. The disease is endemic in parts of Asia and Central America.

Based on all medical information about the 38-year-old patient and his life history in rural Guatemala, physicians identified neurocysticercosis as the most likely cause. Sudden convulsions and brain damage.

After he was first hospitalized, he was given several doses of anticonvulsant, intubated, and moved to the Neuroscience Intensive Care Unit. Once he settled down and left, doctors started treating him with antiparasitic drugs and an anti-inflammatory drug, and continued to use anticonvulsants. He was released from hospital after five days without any neurological symptoms or seizures.

Doctors followed him for three years. After months of treatment, additional brain scans showed that swelling around the largest lesion in the right frontal lobe had decreased. He also remained without seizures, although he was still taking his anticonvulsant medication. It is not known at this time what he will do after leaving the position.

Intestinal parasites have penetrated the human brain and lived there for years
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